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Giovanna Elvira Granato

Researcher at Temple University

Publications -  4
Citations -  5255

Giovanna Elvira Granato is an academic researcher from Temple University. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 3, co-authored 4 publications receiving 4816 citations. Previous affiliations of Giovanna Elvira Granato include University of Naples Federico II.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
Journal ArticleDOI

Bovine herpesvirus type 4 infection modulates autophagy in a permissive cell line.

Serena Montagnaro, +10 more
- 01 Jul 2013 - 
TL;DR: The findings suggest that BoHV‐4 has developed mechanisms for modulation of autophagy that are probably part of a strategy designed to enhance viral replication and to evade the immune system.
Proceedings ArticleDOI

Abstract 1675: Enhanced cell proliferation and induced autophagy in PC-3 prostate cancer cells by 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure.

Giovanna Elvira Granato, +7 more
- 15 Apr 2013 - 
TL;DR: Treatment of PC-3 with TCDD results in enhanced cell proliferation and activation of autophagy, potentiating the transformed phenotype of these cancer cells, and the molecular mechanisms of carcinogenesis of this compound have not been fully investigated.