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Franziska Wild

Researcher at Heidelberg University

Publications -  10
Citations -  5607

Franziska Wild is an academic researcher from Heidelberg University. The author has contributed to research in topics: Autophagy & Neuromuscular junction. The author has an hindex of 8, co-authored 10 publications receiving 5107 citations. Previous affiliations of Franziska Wild include Mannheim University of Applied Sciences & Karlsruhe Institute of Technology.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Sympathetic innervation controls homeostasis of neuromuscular junctions in health and disease

Muzamil Majid Khan, +28 more
- 19 Jan 2016 - 
TL;DR: It is demonstrated that sympathetic neurons make close contact with neuromuscular junctions and form a network in skeletal muscle that may functionally couple different targets including blood vessels, motor neurons, and muscle fibers.
Journal ArticleDOI

Role of autophagy, SQSTM1, SH3GLB1, and TRIM63 in the turnover of nicotinic acetylcholine receptors

Muzamil Majid Khan, +11 more
- 01 Jan 2014 - 
TL;DR: It is suggested that selective autophagy regulates the basal and atrophy-induced turnover of the pentameric transmembrane protein, CHRN, and that TRIM63, together with SH3GLB1 and SQSTM1 regulate this process.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4

Perrine Castets, +19 more
- 18 Jul 2019 - 
TL;DR: It is shown that a balanced activation of mTORC1 contributes to the dynamic regulation of autophagic flux in denervated muscle and that activation of PKB/Akt promotes the nuclear import of HDAC4, which is essential for endplate maintenance upon nerve injury.