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Cam Patterson

Researcher at Cornell University

Publications -  51
Citations -  6173

Cam Patterson is an academic researcher from Cornell University. The author has contributed to research in topics: Ubiquitin ligase & Skeletal muscle. The author has an hindex of 18, co-authored 51 publications receiving 5490 citations. Previous affiliations of Cam Patterson include NewYork–Presbyterian Hospital & University of Göttingen.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Emerging Roles of Vascular Endothelium in Metabolic Homeostasis.

Xinchun Pi, +2 more
- 03 Aug 2018 - 
TL;DR: The concept that the endothelium adjusts its barrier function to control the transendothelial transport of fatty acids, lipoproteins, LPLs (lipoprotein lipases), glucose, and insulin is introduced.
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The role of BMPs in endothelial cell function and dysfunction

Laura A. Dyer, +2 more
- 01 Sep 2014 - 
TL;DR: This review addresses recent advances in the roles of BMP signaling in the endothelium and how BMPs affect endothelial dysfunction and human disease.
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Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
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Depletion of PHD3 protects heart from ischemia/reperfusion injury by inhibiting cardiomyocyte apoptosis.

Liang Xie, +5 more
- 01 Mar 2015 - 
TL;DR: Depletion of PHD3 leads to increased stabilization of HIF-1α and inhibition of DNA damage response, both of which may contribute to the cardioprotective effect seen with depletion ofPHD3.