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Jianjie Ma

Researcher at Ohio State University

Publications -  314
Citations -  19650

Jianjie Ma is an academic researcher from Ohio State University. The author has contributed to research in topics: Skeletal muscle & Ryanodine receptor. The author has an hindex of 67, co-authored 292 publications receiving 17313 citations. Previous affiliations of Jianjie Ma include Rush University Medical Center & University of Medicine and Dentistry of New Jersey.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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NAADP mobilizes calcium from acidic organelles through two-pore channels

TL;DR: It is shown that two-pore channels (TPCs) comprise a family of NAADP receptors, with human TPC1 and chicken TPCN3 being expressed on endosomal membranes, andhuman TPC2 on lysosome membranes when expressed in HEK293 cells, which will advance the understanding of the physiological role ofNAADP.
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Purified ryanodine receptor from rabbit skeletal muscle is the calcium-release channel of sarcoplasmic reticulum.

TL;DR: The ryanodine receptor of rabbit skeletal muscle sarcoplasmic reticulum was purified as a single 450,000-dalton polypeptide from CHAPS- solubilized triads using immunoaffinity chromatography and shows that the purified receptor forms cationic channels selective for divalent ions.
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MG53 nucleates assembly of cell membrane repair machinery

TL;DR: It is reported that MG53, a muscle-specific tripartite motif family protein (TRIM72), is a component of the sarcolemmal membrane-repair machinery and that MG 53 may initiate the assembly of the membrane repair machinery in an oxidation-dependent manner.
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Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells

TL;DR: The results show that mitochondrial depolarization accompanies cytochromec release in pheochromocytoma-6 cells undergoing apoptosis, and that mitochondrial membrane potential decreased abruptly after treatment with staurosporine.