scispace - formally typeset
E

Elio Ziparo

Researcher at Sapienza University of Rome

Publications -  109
Citations -  9803

Elio Ziparo is an academic researcher from Sapienza University of Rome. The author has contributed to research in topics: Sertoli cell & Apoptosis. The author has an hindex of 39, co-authored 109 publications receiving 9006 citations. Previous affiliations of Elio Ziparo include University of Bologna & University of L'Aquila.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Pure Sertoli Cell Cultures: A New Model for the Study of Somatic—Germ Cell Interactions

M. Galdieri, +4 more
- 10 Sep 1981 - 
TL;DR: It was found that the optimal conditions for germ cell detachment without Sertoli cell impairment consist of incubation for 2.5 minutes at 20 C in 20 m TRISHCI.
Journal ArticleDOI

Toll-like receptor 3 triggers apoptosis of human prostate cancer cells through a PKC-α-dependent mechanism

Alessio Paone, +7 more
- 01 Jul 2008 - 
TL;DR: It is shown that a novel interferon-independent pathway involving protein kinase C (PKC)-alpha activation, upstream of p38 and c-jun N-terminal kinase, is responsible for poly (I:C) pro-apoptotic effects on LNCaP cells.
Journal ArticleDOI

Multifaceted Roles of GSK-3 in Cancer and Autophagy-Related Diseases

Romina Mancinelli, +8 more
- 12 Dec 2017 - 
TL;DR: The studies summarized here underline the GSK-3 multifaceted role and indicate such kinase as a molecular target in different pathologies, including diseases associated with autophagy dysregulation.
Journal ArticleDOI

VEGF-induced neoangiogenesis is mediated by NAADP and two-pore channel-2-dependent Ca2+ signaling

Annarita Favia, +11 more
- 04 Nov 2014 - 
TL;DR: It is demonstrated that a VEGFR2/NAADP/TPC2/Ca2+ signaling pathway is critical for VEGF-induced angiogenesis in vitro and in vivo, and that the angiogenic response can be abolished, in cultured cells and in vitro, by inhibiting components of this signaling cascade.