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Michelle A. Ozbun

Researcher at University of New Mexico

Publications -  62
Citations -  8375

Michelle A. Ozbun is an academic researcher from University of New Mexico. The author has contributed to research in topics: Cellular differentiation & HPV infection. The author has an hindex of 32, co-authored 57 publications receiving 7706 citations. Previous affiliations of Michelle A. Ozbun include Foundation University, Islamabad & Pennsylvania State University.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Cellular Entry of Human Papillomavirus Type 16 Involves Activation of the Phosphatidylinositol 3-Kinase/Akt/mTOR Pathway and Inhibition of Autophagy

Zurab Surviladze, +3 more
- 01 Mar 2013 - 
TL;DR: The HPV-host cell interaction stimulates the PI3K/Akt/mTOR pathway and inhibits autophagy, and in combination these events benefit virus infection.
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Synthesis of infectious human papillomavirus type 18 in differentiating epithelium transfected with viral DNA.

Craig Meyers, +2 more
- 01 Oct 1997 - 
TL;DR: HPV18 virions purified by isopycnic gradient were capable of infecting keratinocytes in vitro, as shown by the expression of multiple HPV18-specific, spliced transcripts.
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Characterization of late gene transcripts expressed during vegetative replication of human papillomavirus type 31b.

Michelle A. Ozbun, +1 more
- 01 Jul 1997 - 
TL;DR: The raft system was used to analyze the temporal expression patterns of HPV31b late gene transcripts during the viral life cycle, and characterized transcripts with the potential to encode late gene products.
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Essential roles for soluble virion-associated heparan sulfonated proteoglycans and growth factors in human papillomavirus infections.

Zurab Surviladze, +2 more
- 09 Feb 2012 - 
TL;DR: It is reported that HPV16 particles are not liberated from bound HSPG attachment factors by dissociation, but rather are released by a process previously unreported for pathogen-host cell interactions, which supports an infection model whereby HPV usurps normal host mechanisms for presenting growth factors to cells via soluble H SPG complexes.