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Jane E.B. Reusch

Researcher at Anschutz Medical Campus

Publications -  205
Citations -  14552

Jane E.B. Reusch is an academic researcher from Anschutz Medical Campus. The author has contributed to research in topics: Diabetes mellitus & Type 2 diabetes. The author has an hindex of 52, co-authored 177 publications receiving 13023 citations. Previous affiliations of Jane E.B. Reusch include United States Department of Veterans Affairs & Durham University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Akt/protein kinase B up-regulates Bcl-2 expression through cAMP-response element-binding protein.

Subbiah Pugazhenthi, +8 more
- 14 Apr 2000 - 
TL;DR: Data indicate that regulation of Bcl-2 expression by IGF-I involves a signaling cascade mediated by PI 3-kinase/PDK1/Akt/CREB, and the observation that enhanced CREB activity by Akt signaling leads to increased B cl-2 promoter activity and cell survival.
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CREB Activation Induces Adipogenesis in 3T3-L1 Cells

Jane E.B. Reusch, +2 more
- 01 Feb 2000 - 
TL;DR: The data firmly establish CREB as a primary regulator of adipogenesis and suggest that CREB may play similar roles in other cells and tissues.
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Diabetes and Advanced Glycoxidation End-Products

Amy G. Huebschmann, +3 more
- 01 Jun 2006 - 
TL;DR: The incidence of diabetes, especially type 2 diabetes, is increasing at an alarming rate assuming epidemic proportions (1). Worldwide, 124 million people had diabetes by 1997, although an estimated 221 million people will have diabetes by the year 2010.
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Insulin resistance in adolescents with type 1 diabetes and its relationship to cardiovascular function

Kristen J. Nadeau, +8 more
- 01 Feb 2010 - 
TL;DR: The phenotype of IR in T1D youth was unique, suggesting a pathophysiology that is different from T2D, yet may adversely affect long-term cardiovascular outcomes, and impaired functional exercise capacity and cardiovascular dysfunction.