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Clémence Richetta

Researcher at French Institute of Health and Medical Research

Publications -  15
Citations -  5958

Clémence Richetta is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Biology & Autophagy. The author has an hindex of 7, co-authored 7 publications receiving 5433 citations. Previous affiliations of Clémence Richetta include Pierre-and-Marie-Curie University & University of Lyon.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Autophagy Induction by the Pathogen Receptor CD46

Pierre-Emmanuel Joubert, +24 more
- 22 Oct 2009 - 
TL;DR: It is reported here that the engagement of CD46, a ubiquitous human surface receptor able to bind several different pathogens, is sufficient to induce autophagy, a critical step to control infection.
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IRGM Is a Common Target of RNA Viruses that Subvert the Autophagy Network

Isabel Pombo Grégoire, +35 more
- 08 Dec 2011 - 
TL;DR: This work reveals an unexpected role of IRGM in virus-inducedautophagy and suggests that several different families of RNA viruses may use common strategies to manipulate autophagy to improve viral infectivity.
Journal ArticleDOI

Autophagy in antiviral innate immunity.

Clémence Richetta, +3 more
- 01 Mar 2013 - 
TL;DR: This review highlights recent findings on the cross‐talk between autophagy and innate immunity during viral infections.
Journal ArticleDOI

Sustained autophagy contributes to measles virus infectivity.

Clémence Richetta, +8 more
- 26 Sep 2013 - 
TL;DR: It is reported here that measles virus infection induces successive autophagy signallings in permissive cells, via distinct and uncoupled molecular pathways.