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Maite G. Fernandez-Barrena

Researcher at University of Navarra

Publications -  90
Citations -  8980

Maite G. Fernandez-Barrena is an academic researcher from University of Navarra. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 24, co-authored 65 publications receiving 7529 citations. Previous affiliations of Maite G. Fernandez-Barrena include Mayo Clinic & Carlos III Health Institute.

Papers
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Stromal elements act to restrain, rather than support, pancreatic ductal adenocarcinoma

Andrew D. Rhim, +17 more
- 16 Jun 2014 - 
TL;DR: De deleting Shh in a well-defined mouse model of PDAC demonstrated that some components of the tumor stroma can act to restrain tumor growth, and administration of VEGFR blocking antibody selectively improved survival of Shh-deficient tumors.
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Identification of fibroblast growth factor 15 as a novel mediator of liver regeneration and its application in the prevention of post-resection liver failure in mice

Iker Uriarte, +14 more
- 01 Jun 2013 - 
TL;DR: Fgf15 is necessary to maintain BA homeostasis and prevent liver injury during liver regeneration and is an essential mediator of the liver growth-promoting effects of BA.
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Fibroblast growth factor 15/19 (FGF15/19) protects from diet-induced hepatic steatosis: development of an FGF19-based chimeric molecule to promote fatty liver regeneration.

Gloria Alvarez-Sola, +21 more
- 01 Oct 2017 - 
TL;DR: FGF15/19 is essential for hepatic metabolic adaptation to dietary fat being a physiological regulator of Pparγ2 expression and perioperative administration of Fibapo improves fatty liver regeneration.
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Galectin-1 Drives Pancreatic Carcinogenesis through Stroma Remodeling and Hedgehog Signaling Activation

Neus Martínez-Bosch, +13 more
- 01 Jul 2014 - 
TL;DR: Mechanistic investigations revealed that Gal1 promoted Hedgehog pathway signaling in PDAC cells and stromal fibroblasts as well as in Ela-myc tumors, establishing a function for Gal1 in tumor-stroma crosstalk inPDAC and providing a preclinical rationale for Gal 1 targeting as a microenvironment-based therapeutic strategy.