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Xiaonan Zhao

Researcher at Shanghai Jiao Tong University

Publications -  15
Citations -  5448

Xiaonan Zhao is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 7, co-authored 9 publications receiving 4979 citations.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Regulation of autophagy by E3 ubiquitin ligase RNF216 through BECN1 ubiquitination.

TL;DR: This study finds that RNF216 (ring finger protein 216), an E3 ubiquitin ligase, strongly inhibits autophagy in macrophages and suggests that R NF216 may be a target for treatment of inflammatory diseases.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
Journal ArticleDOI

Adenylyl Cyclase 6 Activation Negatively Regulates TLR4 Signaling through Lipid Raft–Mediated Endocytosis

TL;DR: It is demonstrated that adenylyl cyclase (AC) activation attenuates TLR4 signaling in a murine macrophage cell line and bone marrow–derived macrophages when stimulated with LPS, and that the AC6 isoform plays a key role in negative regulation of TLR 4 signaling by promoting protein degradation.