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Gustavo A. Nader

Researcher at Pennsylvania State University

Publications -  53
Citations -  8428

Gustavo A. Nader is an academic researcher from Pennsylvania State University. The author has contributed to research in topics: Skeletal muscle & Muscle hypertrophy. The author has an hindex of 28, co-authored 48 publications receiving 7660 citations. Previous affiliations of Gustavo A. Nader include Children's National Medical Center & Penn State Cancer Institute.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Intracellular signaling specificity in skeletal muscle in response to different modes of exercise

Gustavo A. Nader, +1 more
- 01 May 2001 - 
TL;DR: It is demonstrated that different exercise protocols resulted in the selective activation of specific intracellular signaling pathways, which may determine the specific adaptations induced by different forms of exercise.
Journal ArticleDOI

Concurrent strength and endurance training: from molecules to man.

Gustavo A. Nader
- 01 Nov 2006 - 
TL;DR: There seems to be an explanation for the interference of strength development during concurrent training; it is now clear that different forms of exercise induce antagonistic intracellular signaling mechanisms that, in turn, could have a negative impact on the muscle's adaptive response to this particular form of training.
Journal ArticleDOI

Loss of emerin at the nuclear envelope disrupts the Rb1/E2F and MyoD pathways during muscle regeneration.

Gisela Melcon, +14 more
- 15 Feb 2006 - 
TL;DR: The derivation of mice lacking emerin is described in an attempt to derive a mouse model for EDMD1, suggesting that the dominant LMNA mutations seen in many clinically disparate laminopathies may similarly alter Rb function, with regard to either the timing of exit from the cell cycle or terminal differentiation programs.
Journal ArticleDOI

Molecular determinants of skeletal muscle mass: Getting the "AKT" together

Gustavo A. Nader
- 01 Oct 2005 - 
TL;DR: Recent findings are discussed demonstrating that the AKT1/FOXOs/Atrogin-1(MAFbx)/MuRF1 signaling network plays an important role in the progression of skeletal muscle atrophy.