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Hongbing Zhang

Researcher at Peking University

Publications -  12
Citations -  6039

Hongbing Zhang is an academic researcher from Peking University. The author has contributed to research in topics: Chemistry & Medicine. The author has an hindex of 8, co-authored 8 publications receiving 5589 citations. Previous affiliations of Hongbing Zhang include Peking Union Medical College & University of Texas MD Anderson Cancer Center.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

BRCA1 physically associates with p53 and stimulates its transcriptional activity

Hongbing Zhang, +7 more
- 02 Apr 1998 - 
TL;DR: BRCA1 increases p53-dependent transcription from the p21WAF1/CIP1 and bax promoters and interacts with p53 proteins both in vitro and in vivo, indicating that BRCA 1 and p53 may coordinately regulate gene expression in their role as tumor suppressors.
Journal ArticleDOI

BRCA1, BRCA2, and DNA damage response: collision or collusion?

Hongbing Zhang, +2 more
- 20 Feb 1998 - 
TL;DR: The intriguing finding described by Patel et al. 1998 is the spontaneous occurrence of gross chromosomal abnormalities, which suggests that there is a fundamental defect in chromosomal replication, repair, and/or segregation in BRCA2Tr/Tr cells.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

BRCA1 physically and functionally interacts with ATF1.

Yariv Houvras, +5 more
- 17 Nov 2000 - 
TL;DR: It is demonstrated that BRCA1 and ATF1 can physically associate in vitro, in yeast, and in human cells, and implicate BRCa1 in transcriptional activation of ATF1 target genes, some of which are involved in the transcriptional response to DNA damage.