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Yukitoshi Nagahara

Researcher at Tokyo Denki University

Publications -  43
Citations -  6048

Yukitoshi Nagahara is an academic researcher from Tokyo Denki University. The author has contributed to research in topics: Apoptosis & Jurkat cells. The author has an hindex of 15, co-authored 40 publications receiving 5537 citations. Previous affiliations of Yukitoshi Nagahara include Tokyo University of Science.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

A novel immunosuppressive agent FTY720 induced Akt dephosphorylation in leukemia cells

Yumiko Matsuoka, +3 more
- 01 Apr 2003 - 
TL;DR: The results suggest that FTY720 activated PP2A orPP2A‐like phosphatase and dephosphorylated Akt pathway factors resulting in the enhancement of apoptosis via mitochondria.
Journal ArticleDOI

Immunosuppressant FTY720 induces apoptosis by direct induction of permeability transition and release of cytochrome c from mitochondria.

Yukitoshi Nagahara, +2 more
- 15 Sep 2000 - 
TL;DR: Results indicate that FTY720 directly affects mitochondria and triggers permeability transition to induce further apoptotic events and directly influences mitochondrial functions.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

Evidence that FTY720 induces T cell apoptosis in vivo.

Yukitoshi Nagahara, +4 more
- 01 Jun 2000 - 
TL;DR: It was concluded that apoptosis of blood lymphocytes was induced immediately after administration of FTY720, and the cells could be immediately scavenged by phagocytes or the reticuloendothelial system in addition to Peyer's patches homing.