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Sara Calatayud

Researcher at University of Valencia

Publications -  85
Citations -  7066

Sara Calatayud is an academic researcher from University of Valencia. The author has contributed to research in topics: Nitric oxide & Internal medicine. The author has an hindex of 24, co-authored 71 publications receiving 6300 citations. Previous affiliations of Sara Calatayud include Imperial College London & University of London.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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The activation of Wnt signaling by a STAT6-dependent macrophage phenotype promotes mucosal repair in murine IBD.

TL;DR: It is demonstrated that a STAT6-dependent macrophage phenotype promotes mucosal repair in TNBS-treated mice through activation of the Wnt signaling pathway.
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M2 Macrophages Activate WNT Signaling Pathway in Epithelial Cells: Relevance in Ulcerative Colitis

TL;DR: It is shown that M2 macrophages, and not M1, activate Wnt signalling pathways and decrease enterocyte differentiation in co-cultured epithelial cells in patients with UC.
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Nitric oxide: relation to integrity, injury, and healing of the gastric mucosa.

TL;DR: iNOS activity may be associated to reduced leukocyte‐endothelium interaction and platelet aggregation as well as protection of mucosal microcirculation and may be important to resolve inflammation by increasing apoptosis in inflammatory cells.
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Succinate receptor mediates intestinal inflammation and fibrosis.

TL;DR: It is shown that both serum and intestinal succinate levels and SUCNR1 expression in intestinal surgical resections were higher in Crohn's disease patients than in controls and a role for S UCNR1 in murine intestinal inflammation and fibrosis is shown.