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Clara I. Rodríguez

Researcher at National Institutes of Health

Publications -  39
Citations -  6395

Clara I. Rodríguez is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Mesenchymal stem cell & Stem cell. The author has an hindex of 18, co-authored 36 publications receiving 5685 citations. Previous affiliations of Clara I. Rodríguez include Autonomous University of Madrid & Spanish National Research Council.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Osteogenesis and aging: lessons from mesenchymal stem cells.

Arantza Infante, +1 more
- 26 Sep 2018 - 
TL;DR: This review will focus on the most recent/relevant molecular findings driving this functional impairment of mesenchymal stem cells in the aging process.
Journal ArticleDOI

African Swine Fever Virus IAP Homologue Inhibits Caspase Activation and Promotes Cell Survival in Mammalian Cells

Maria L. Nogal, +6 more
- 15 Mar 2001 - 
TL;DR: It is shown that the viral IAP modulates the proteolytic processing of the effector cell death protease caspase-3 and the apoptosis which are induced in the infected cells and could indicate a conserved mechanism of action for ASFV IAP and other IAP family members to suppress apoptosis.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

Dominant T- and B-cell epitopes in an autoantigen linked to Chagas' disease.

Núria Gironès, +5 more
- 15 Apr 2001 - 
TL;DR: The results demonstrate the presence of both T- and B-cell cross-reactive epitopes in the Cha antigen, which may lead to T/B cell cooperation and give rise to a pathological immunodominant response against Cha in T. cruzi infected animals.