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Panagiotis D. Velentzas

Researcher at University of Massachusetts Medical School

Publications -  17
Citations -  5498

Panagiotis D. Velentzas is an academic researcher from University of Massachusetts Medical School. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 10, co-authored 17 publications receiving 5011 citations. Previous affiliations of Panagiotis D. Velentzas include National and Kapodistrian University of Athens.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
Journal ArticleDOI

The NF-κB Factor Relish Regulates Atg1 Expression and Controls Autophagy.

Anubhab Nandy, +5 more
- 20 Nov 2018 - 
TL;DR: It is shown that the nuclear factor κB (NF-κB) factor Relish, a component of the immune deficiency (Imd) pathway, is required for salivary gland degradation and regulated autophagy during developmentally programmed cell death.
Journal ArticleDOI

Detrimental effects of proteasome inhibition activity in Drosophila melanogaster : implication of ER stress, autophagy, and apoptosis

Panagiotis D. Velentzas, +6 more
- 01 Feb 2013 - 
TL;DR: It is reported that proteasome inhibition drastically shortens fly life-span and impairs climbing performance, while it also causes larval lethality and activates developmentally irregular cell death programs during oogenesis, and proposed that the observed upregulation of autophagy operates as a protective mechanism to “counterbalance” Bortezomib-induced systemic toxicity.