scispace - formally typeset
T

Truong Lam

Researcher at University of Texas Health Science Center at Houston

Publications -  10
Citations -  5432

Truong Lam is an academic researcher from University of Texas Health Science Center at Houston. The author has contributed to research in topics: Cancer research & Medicine. The author has an hindex of 6, co-authored 7 publications receiving 4973 citations. Previous affiliations of Truong Lam include University of Texas at Austin.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
Journal ArticleDOI

Reversal of intramyocellular lipid accumulation by lipophagy and a p62-mediated pathway

TL;DR: It is concluded that lipophagy promotes the clearance of lipids from myocytes and switches to an alternative, p62-mediated, lysosomal-independent pathway in the context of chronic lipid overload.
Journal ArticleDOI

Insulin resistance improves metabolic and contractile efficiency in stressed rat heart

TL;DR: The results suggest that insulin resistance lessens fuel toxicity in the stressed heart and calls for a new exploration of the mechanisms regulating substrate uptake and oxidation in the insulin‐resistant heart.