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Olatz Pampliega

Researcher at University of the Basque Country

Publications -  25
Citations -  6627

Olatz Pampliega is an academic researcher from University of the Basque Country. The author has contributed to research in topics: Autophagy & Cilium. The author has an hindex of 13, co-authored 20 publications receiving 5867 citations. Previous affiliations of Olatz Pampliega include University of Bordeaux & Albert Einstein College of Medicine.

Papers
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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Functional interaction between autophagy and ciliogenesis

TL;DR: This work shows that part of the molecular machinery involved in ciliogenesis also participates in the early steps of the autophagic process, and proposes that basal autophagy regulates ciliary growth through the degradation of proteins required for intraflagellar transport.
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Selective autophagy as a potential therapeutic target for neurodegenerative disorders.

TL;DR: A change of strategy in the modulation of autophagy might hold promise for future disease-modifying therapies for patients with neurodegenerative disorders.
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P2X7 receptors mediate ischemic damage to oligodendrocytes

TL;DR: Data indicate that ATP released during ischemia and the subsequent activation of P2X7 receptor is critical to white matter demise during stroke and point to this receptor type as a therapeutic target to limit tissue damage in cerebrovascular diseases.
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Increased expression of cystine/glutamate antiporter in multiple sclerosis

TL;DR: It is shown that human activated monocytes release glutamate through cystine/glutamate antiporter xc- and that the expression of the catalytic subunit xCT is upregulated as a consequence of monocyte activation, revealing a link between inflammation and excitotoxicity in demyelinating diseases.