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Jian Zhang

Researcher at Nanjing Forestry University

Publications -  74
Citations -  7125

Jian Zhang is an academic researcher from Nanjing Forestry University. The author has contributed to research in topics: Innate lymphoid cell & Interleukin 33. The author has an hindex of 20, co-authored 62 publications receiving 6032 citations. Previous affiliations of Jian Zhang include University of Macau & Jinan University.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Testosterone Attenuates Group 2 Innate Lymphoid Cell-Mediated Airway Inflammation.

TL;DR: Findings provide a foundational understanding of sexual dimorphism in ILC2 function, and testosterone attenuated Alternaria-extract-induced IL-5+ and IL-13+ I LC2 numbers and lung eosinophils by intrinsically decreasing lung ILC 2 numbers.
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Multistructured Electrospun Nanofibers for Air Filtration: A Review.

TL;DR: The preparation methods of high-performance electrospun air filTration membranes (EAFMs) and the typical surface structures and filtration principles of electrospinning nanofibers for air Filtration are reviewed.
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IL-33 promotes the egress of group 2 innate lymphoid cells from the bone marrow

TL;DR: In this article, the authors identified a critical role for IL-33, a hallmark peripheral ILC2-activating cytokine, in promoting the egress of group 2 innate lymphoid cells (ILC2s) from the bone marrow.
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Involvement of nitric oxide released from microglia-macrophages in pathological changes of cathepsin D-deficient mice.

TL;DR: The results suggest that NO production via iNOS activity in microglia and peripheral macrophages contributes to secondary tissue damages such as neuronal apoptosis and intestinal necrosis, respectively.