Thomas Weide

TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.

TL;DR: It is suggested that coordinated action between Rab6a, BICD and the dynein–dynactin complex controls COPI-independent Golgi–ER transport.

TL;DR: It is demonstrated that angiotensin II (Ang II) caused a reactive oxygen species (ROS)-dependent rearrangement of cortical F-actin and a migratory phenotype switch in cultured mouse podocytes with stable Ang II type 1 receptor (AT1R) expression.

TL;DR: The data provide new molecular insights explaining which pathways and molecules are affected in podocytes by an imbalanced mTOR function because of rapamycin treatment, and reduce cell adhesion and cell motility, which was accompanied by an enhanced formation of dot-like actin-rich structures.

TL;DR: It is reported for the first time that the rab1b protein interacts specificially with GM130 in a GTP‐dependent manner and therefore needs the hypervariable regions of the N‐ and C‐termini.