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Glauber Costa Brito

Researcher at University of São Paulo

Publications -  13
Citations -  5697

Glauber Costa Brito is an academic researcher from University of São Paulo. The author has contributed to research in topics: Membrane protein & Gene. The author has an hindex of 10, co-authored 13 publications receiving 5202 citations. Previous affiliations of Glauber Costa Brito include Sunnybrook Research Institute & Universidade Federal da Grande Dourados.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Antisense intronic non-coding RNA levels correlate to the degree of tumor differentiation in prostate cancer.

Eduardo M. Reis, +25 more
- 26 Aug 2004 - 
TL;DR: The construction and use of a cDNA microarray platform enriched in intronic transcripts to assess their biological relevance in pathological conditions and the correlation between the low/high degrees of tumor differentiation and antisense intronic RASSF1 transcript levels is confirmed.
Journal Article

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2459 more
- 01 Jan 2016 - 
Journal ArticleDOI

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Daniel J. Klionsky, +2522 more
- 01 Jan 2016 - 
TL;DR: Author(s): Klionsky, DJ; Abdelmohsen, K; Abe, A; Abedin, MJ; Abeliovich, H; A Frozena, AA; Adachi, H, Adeli, K, Adhihetty, PJ; Adler, SG; Agam, G; Agarwal, R; Aghi, MK; Agnello, M; Agostinis, P; Aguilar, PV; Aguirre-Ghis
Journal ArticleDOI

Phosphorylation switches Bax from promoting to inhibiting apoptosis thereby increasing drug resistance.

Justin Kale, +7 more
- 09 Jul 2018 - 
TL;DR: Increased phosphorylated anti‐apoptotic Bax promotes resistance of cancer cells to inherent and drug‐induced apoptosis, and analysis of the TCGA database reveals that 98% of cancer patients with increased BAX levels also have an upregulated Akt pathway.