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Hing Y. Leung

Researcher at University of Glasgow

Publications -  238
Citations -  14795

Hing Y. Leung is an academic researcher from University of Glasgow. The author has contributed to research in topics: Prostate cancer & Cancer. The author has an hindex of 56, co-authored 217 publications receiving 13211 citations. Previous affiliations of Hing Y. Leung include National Health Service & University of Newcastle.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Upregulation and Nuclear Recruitment of HDAC1 in Hormone Refractory Prostate Cancer

Kalipso Halkidou, +5 more
- 01 May 2004 - 
TL;DR: Histone deacetylase 1 (HDAC1) is a co‐repressor involved in differentiation and proliferation control and targets a number of transcription factors including p53.
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Expression of Tip60, an androgen receptor coactivator, and its role in prostate cancer development.

Kalipso Halkidou, +9 more
- 24 Apr 2003 - 
TL;DR: In vitro and in vivo data support a possible role for Tip60 in the molecular pathway leading to the development of androgen-independent CaP following long-term androgen deprivation therapy.
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Expression of RAC 3, a steroid hormone receptor co-activator in prostate cancer

Vincent J. Gnanapragasam, +4 more
- 14 Dec 2001 - 
TL;DR: It is concluded that RAC 3 is an important co-activator of the AR in the prostate and may have an important role in the progression of prostate cancer.
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The RNA helicase p68 is a novel androgen receptor coactivator involved in splicing and is overexpressed in prostate cancer.

Emma L. Clark, +11 more
- 01 Oct 2008 - 
TL;DR: Findings implicate p68 as a novel AR transcriptional coactivator that is significantly overexpressed in PCa with a possible role in progression to hormone-refractory disease.