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Anita Solhaug

Researcher at National Veterinary Institute

Publications -  38
Citations -  6510

Anita Solhaug is an academic researcher from National Veterinary Institute. The author has contributed to research in topics: Apoptosis & DNA damage. The author has an hindex of 22, co-authored 33 publications receiving 5904 citations. Previous affiliations of Anita Solhaug include University of Oslo & Norwegian Institute of Public Health.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Polycyclic aromatic hydrocarbons induce both apoptotic and anti-apoptotic signals in Hepa1c1c7 cells.

Anita Solhaug, +5 more
- 19 Dec 2003 - 
TL;DR: B[a]P and the CP-PAH induced apoptotic as well as anti-apoptotic signals in Hepa1c1c7 cells and neither of the compounds triggered apoptosis in primary cultures of rat lung cells, possibly due to a lack of CYP1A1 induction.
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Effects of nitrated-polycyclic aromatic hydrocarbons and diesel exhaust particle extracts on cell signalling related to apoptosis: Possible implications for their mutagenic and carcinogenic effects

Nina E. Landvik, +6 more
- 07 Mar 2007 - 
TL;DR: The most mutagenic and carcinogenic compound tested, 1,8-DNP, induced little cell death, despite the fact that this compound seemed to give the most DNA damage as judged by DNA adduct formation.
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Mechanisms of Action and Toxicity of the Mycotoxin Alternariol: A Review

Anita Solhaug, +2 more
- 01 Dec 2016 - 
TL;DR: In vitro mechanistic studies further support the in vivo studies suggesting low acute toxicity of AOH, and suggest a decreased immune response to infections and/or a disturbed balance of the adaptive immune system when exposed together with other mycotoxins cannot be excluded.
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Different mechanisms involved in apoptosis following exposure to benzo[a]pyrene in F258 and Hepa1c1c7 cells.

Jørn A. Holme, +9 more
- 05 Apr 2007 - 
TL;DR: The results suggest that the rate of metabolism of B[a]P and type of reactive metabolites formed influence the resulting balance of pro-APoptotic and anti-apoptotic cell signaling, and hence the mechanisms involved in cell death and the chances of more permanent genetic damage.