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Vania Gelmetti

Researcher at Casa Sollievo della Sofferenza

Publications -  37
Citations -  11213

Vania Gelmetti is an academic researcher from Casa Sollievo della Sofferenza. The author has contributed to research in topics: Myeloid leukemia & PINK1. The author has an hindex of 29, co-authored 36 publications receiving 10533 citations. Previous affiliations of Vania Gelmetti include University of Perugia & University of Rome Tor Vergata.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Fusion proteins of the retinoic acid receptor-alpha recruit histone deacetylase in promyelocytic leukaemia.

Fausto Grignani, +13 more
- 19 Feb 1998 - 
TL;DR: It is shown that both PML–RARα and PLZF–Rarα fusion proteins recruit the nuclear co-repressor (N-CoR)–histone deacetylase complex through the RARα CoR box, which determines the differential response of APLs to RA.
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A Minicircuitry Comprised of MicroRNA-223 and Transcription Factors NFI-A and C/EBPα Regulates Human Granulopoiesis

Francesco Fazi, +6 more
- 02 Dec 2005 - 
TL;DR: The data indicate that miR-223 plays a crucial role during granulopoiesis and point to the NFI-A repression as an important molecular pathway mediating gene reprogramming in this cell lineage.
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Inhibitors of histone deacetylases induce tumor-selective apoptosis through activation of the death receptor pathway.

Alessandra Insinga, +9 more
- 01 Jan 2005 - 
TL;DR: Results show that sensitivity to HDACIs in leukemias is a property of the fully transformed phenotype and depends on activation of a specific death pathway.
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Aberrant Recruitment of the Nuclear Receptor Corepressor-Histone Deacetylase Complex by the Acute Myeloid Leukemia Fusion Partner ETO

Vania Gelmetti, +5 more
- 01 Dec 1998 - 
TL;DR: It is reported that ETO, which is fused to the acute myelogenous leukemia 1 (AML1) transcription factor in t(8;21) AML, interacts via its zinc finger region with a conserved domain of the corepressors N-CoR and SMRT and recruits HDAC in vivo.