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Michael T. Greenwood

Researcher at Royal Military College of Canada

Publications -  57
Citations -  8503

Michael T. Greenwood is an academic researcher from Royal Military College of Canada. The author has contributed to research in topics: Programmed cell death & G protein-coupled receptor. The author has an hindex of 29, co-authored 57 publications receiving 7867 citations. Previous affiliations of Michael T. Greenwood include McGill University & Concordia University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Anti-apoptosis and cell survival: A review

Liam Portt, +4 more
- 01 Jan 2011 - 
TL;DR: This work discusses the different strategies that are used to prevent cell death and illustrates that although anti-apoptosis and cellular survival serve to counteract PCD, they are nevertheless mechanistically distinct from the processes that regulate cell death.
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The somatostatin receptor family

Yogesh C. Patel, +5 more
- 18 Aug 1995 - 
TL;DR: Structural-function studies indicate that the core residues in SST-14 ligand Phe6-Phe11 dock within a ligand binding pocket located in TMDs 3-7 which is lined by hydrophobic and charged amino acid residues.
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All Five Cloned Human Somatostatin Receptors (hSSTR1-5) Are Functionally Coupled to Adenylyl Cyclase

Yogesh C. Patel, +4 more
- 31 Jan 1994 - 
TL;DR: All 5 human SSTRs are functionally coupled to inhibition of adenylyl cyclase in CHO-K1 cells via pertussis toxin sensitive G proteins, demonstrating their binding affinities for the individual hSSTR subtypes.
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Iron mediated toxicity and programmed cell death: A review and a re-examination of existing paradigms.

Rawan Eid, +2 more
- 01 Feb 2017 - 
TL;DR: It is shown that existing regulatory processes are more than adequate to limit the toxicity of iron even in response to iron overload and that intracellular iron is not toxic but a stress responsive programmed cell death-inducing second messenger.