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Soledad Matus

Researcher at University of Chile

Publications -  36
Citations -  7819

Soledad Matus is an academic researcher from University of Chile. The author has contributed to research in topics: Unfolded protein response & Endoplasmic reticulum. The author has an hindex of 22, co-authored 34 publications receiving 7082 citations. Previous affiliations of Soledad Matus include Pontifical Catholic University of Chile & San Sebastián University.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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XBP-1 deficiency in the nervous system protects against amyotrophic lateral sclerosis by increasing autophagy

Claudio Hetz, +9 more
- 01 Oct 2009 - 
TL;DR: The results reveal a new function of XBP-1 in the control of autophagy and indicate critical cross-talk between these two signaling pathways that can provide protection against neurodegeneration.
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Trehalose delays the progression of amyotrophic lateral sclerosis by enhancing autophagy in motoneurons.

Karen Castillo, +9 more
- 06 Jun 2013 - 
TL;DR: Trehalose treatment led to a significant upregulation in the expression of key autophagy-related genes at the mRNA level including Lc3, Becn1, Sqstm1 and Atg5, and trehalose administration enhanced the nuclear translocation of FOXO1, an important transcription factor involved in the activation of autophagic in neurons.
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Protein folding stress in neurodegenerative diseases: a glimpse into the ER.

Soledad Matus, +3 more
- 01 Apr 2011 - 
TL;DR: The particular mechanisms currently proposed to be involved in the generation of protein folding stress in different neurodegenerative conditions are discussed and possible therapeutic interventions are speculated about.
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Antiribosomal-P autoantibodies from psychiatric lupus target a novel neuronal surface protein causing calcium influx and apoptosis.

Soledad Matus, +10 more
- 24 Dec 2007 - 
TL;DR: It is shown that anti-P antibodies recognize a new integral membrane protein of the neuronal cell surface that is preferentially distributed in areas involved in memory, cognition, and emotion and provides a molecular target for future exploration of this and other psychiatric diseases.