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Justine D. Mintern

Researcher at University of Melbourne

Publications -  106
Citations -  9792

Justine D. Mintern is an academic researcher from University of Melbourne. The author has contributed to research in topics: Antigen presentation & Antigen. The author has an hindex of 37, co-authored 95 publications receiving 8695 citations. Previous affiliations of Justine D. Mintern include QIMR Berghofer Medical Research Institute & Walter and Eliza Hall Institute of Medical Research.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Initiation of Autoimmune Diabetes by Developmentally Regulated Presentation of Islet Cell Antigens in the Pancreatic Lymph Nodes

TL;DR: It is concluded that beta cell antigens are transported specifically to pancreatic lymph nodes, where they trigger reactive T cells to invade the islets.
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Enhanced survival of lung tissue-resident memory CD8+ T cells during infection with influenza virus due to selective expression of IFITM3

TL;DR: It is found that lung TRM cells selectively maintained expression of the interferon-induced transmembrane protein IFITM3, a protein that confers broad resistance to viral infection.
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Blood-stage Plasmodium infection induces CD8+ T lymphocytes to parasite-expressed antigens, largely regulated by CD8α+ dendritic cells

TL;DR: It is shown that blood-stage infection leads to parasite-specific CD8+ and CD4+ T cell responses, and that P. berghei-expressed antigens are cross-presented by the CD8α+ subset of dendritic cells (DC), and that this induces pathogen-specific cytotoxic T lymphocytes (CTL) capable of lysing cells presenting antIGens expressed by blood- stage parasites.