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Arnaud Moris

Researcher at University of Paris

Publications -  68
Citations -  9756

Arnaud Moris is an academic researcher from University of Paris. The author has contributed to research in topics: Cytotoxic T cell & Immune system. The author has an hindex of 33, co-authored 65 publications receiving 8993 citations. Previous affiliations of Arnaud Moris include Université Paris-Saclay & University of Tübingen.

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Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Infusion of cytomegalovirus (CMV)-specific T cells for the treatment of CMV infection not responding to antiviral chemotherapy.

Hermann Einsele, +13 more
- 01 Jun 2002 - 
TL;DR: Anti-CMV cellular therapy was successful in 5 of 7 patients, whereas in 2 of7 patients, who received an intensified immune suppression at the time of or after T-cell therapy, only transient reductions in virus load were obtained.
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Human immunodeficiency virus-1 inhibition of immunoamphisomes in dendritic cells impairs early innate and adaptive immune responses.

Fabien Blanchet, +13 more
- 28 May 2010 - 
TL;DR: The findings that HIV-1 downregulates autophagy and impedes immune functions of DCs represent a pathogenesis mechanism that can be pharmacologically countered with therapeutic and prophylactic implications.
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Antigen sensitivity is a major determinant of CD8+ T-cell polyfunctionality and HIV-suppressive activity

Jorge R. Almeida, +12 more
- 18 Jun 2009 - 
TL;DR: It is reported that attributes of CD8(+) T-cell efficacy against HIV are linked at the level of antigen sensitivity, and vaccine strategies should focus on the induction of HIV-specific T cells with high levels of antigenensitivity to elicit potent antiviral efficacy.
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Activation of the lectin DC-SIGN induces an immature dendritic cell phenotype triggering Rho-GTPase activity required for HIV-1 replication

Ashleigh Hodges, +10 more
- 01 Jun 2007 - 
TL;DR: HIV sequestration by and stimulation of DC-SIGN helps HIV evade immune responses and spread to cells by mediated by the Rho guanine nucleotide–exchange factor LARG and led to increased Rho-GTPase activity.