scispace - formally typeset
Y

Yong Xia

Researcher at Guangzhou Medical University

Publications -  17
Citations -  7855

Yong Xia is an academic researcher from Guangzhou Medical University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 8, co-authored 9 publications receiving 7302 citations. Previous affiliations of Yong Xia include Johns Hopkins University & Ohio State University.

Papers
More filters
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Mitogenic Signaling Mediated by Oxidants in Ras-Transformed Fibroblasts

Kaikobad Irani, +8 more
- 14 Mar 1997 - 
TL;DR: H-RasV12-induced transformation can lead to the production of ·O2− through one or more pathways involving a flavoprotein and Rac1, suggesting a possible mechanism for the effects of antioxidants against Ras-induced cellular transformation.
Journal ArticleDOI

Nitric oxide synthase generates superoxide and nitric oxide in arginine-depleted cells leading to peroxynitrite-mediated cellular injury

Yong Xia, +4 more
- 25 Jun 1996 - 
TL;DR: With reduced L-Arg availability NOS elicits cytotoxicity by generating .O2- and NO that interact to form the potent oxidant peroxynitrite and regulating arginine levels may provide a therapeutic approach to disorders involving .O 2-/NO-mediated cellular injury.
Journal ArticleDOI

Oxygen radical-mediated reduction in basal and agonist-evoked NO release in isolated rat heart.

Nazareno Paolocci, +10 more
- 01 Apr 2001 - 
TL;DR: OfR severely impair NO-mediated coronary vasorelaxation affecting both basal and agonist-evoked NO release but not eNOS activity, however, EL also significantly blunts NOS/COX-independent vasodilation suggesting alteration of other vasodilatative pathways.
Journal ArticleDOI

Adverse ventricular remodeling and exacerbated NOS uncoupling from pressure-overload in mice lacking the beta3-adrenoreceptor.

An L. Moens, +21 more
- 01 Nov 2009 - 
TL;DR: A cardioprotective role for the beta3-AR is suggested in modulating oxidative stress and adverse remodeling in the failing heart, all of which can be rescued with exogenous BH4.